Although a few TK genes have been shown to be mutationally altered in specific human cancers (1), it is not known how many or how often members of the TK gene family are altered in any particular cancer type.

盡管有少數(shù)TK基因被報(bào)道在特定的人類腫瘤中有突變，但對(duì)整個(gè)TK基因家族中有多少個(gè)基因多少次在某一特定腫瘤類型中有突變久久九九这里只有精品久久久久，還是個(gè)未知數(shù)。

Thus, IDH1 appears to function as a tumor suppressor that, when mutationally inactivated, contributes to tumorigenesis in part through induction of the HIF-1 pathway.

因而IDH1似乎是作為一種腫瘤抑制因子而存在国产人人干人人射，一旦通過突變使之滅活亚洲强奸，則可能會(huì)部分的通過HIF-1α途徑參與腫瘤的發(fā)生。

Subsequent studies of mutationally altered AMPK activity explained the dominant mode of disease inheritance and the phenotypes of cardiac glycogen accumulation and preexcitation in the conduction system.

隨后Www.视频一区二区，PRKAG2突變改變AMPK活性的研究解釋了疾病顯性遺傳的模式中文字幕韩国三级理论电影、心臟糖原積累及傳導(dǎo)系統(tǒng)中預(yù)激的表現(xiàn)型。

Thus, IDH1 appears to function as a tumor suppressor that, when mutationally inactivated, contributes to tumorigenesis in part through induction of the HIF-1 pathway.

因而異檸檬酸脫氫酶-1(IDH1)似乎是作為一種腫瘤抑制因子而存在日韩系列在线精品视频免费观看，一旦通過突變使之滅活天堂黄色网站在线，則可能會(huì)部分的通過缺氧誘導(dǎo)因子亞單位α（HIF-1α）的途徑而參與腫瘤的發(fā)生。

Thus, IDH1 appears to function as a tumor suppressor that, when mutationally inactivated, contributes to tumorigenesis in part through induction of the HIF-1 pathway.

研究者認(rèn)為日本最新一区二区三区四区在线播放，IDH1具有腫瘤抑制子的作用，一旦IDH1發(fā)生突變失活則導(dǎo)致HIF-1通路發(fā)生改變促進(jìn)腫瘤發(fā)生。