Results:(1) Progesterone-treated models showed intact nasal mucosa, regular ciliary lining and inactive glands;(2) Untreated or normal saline treated models showed disrupted mucosa, inverted cilia and massive mucosal infiltration of neutrophils;(3) Smearing of nasal discharge revealed limited vs abundant number of neutrophils in SD models treated with progesterone vs untreated or treated with normal saline.(4) Hoechst stain: significantly fewer apoptotic cells per field were found in progesterone-treated models (1.583 ± 0.28) compared with untreated or normal saline treated models (2.85 ± 0.285 and 4.8 ± 0.715, respectively).

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The results show that: This tablet can decrease the rat"s footpad swell induced by Freud"s, inhibit the permeability of the blood capillary in inflammatory joint and decrease the 11-2 level in serum while increasing the rat"s body weight,(2) It can lower the rat"s temperature and the permeability of the blood capillary in inflammatory joint induced by collagen and inhibit the delayed type hypersensitivity,It can lower the stickiness of full blood and the accumulation of erythrocyte obviously inblood stasis model rats,lt can inhibit the formation of hemolysin and decrease the index of carbon granule clearance in mice, It can lower the permeability of celiac blood capillary and inhibit the granulomatous hyperplasia by sc cotton ,It can inhibit the pain induced by acetic acid and raise the pain threshold value of heat stimulation.

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To choose SD rat as experiment obiect, which was divided in to 5 groups: normal control group, model group, methylaminopterin-treat group, tripterygiumglycosides-treat group, and GuiZhiShaoYaoZhiMuTang-treat group, establishing immunityinflammation rat model., through measuring rat voix pedis intumesce degree, the content ofIL-6, TNF-A in peripheral blood, and rats weight as well as organ index, we can observe theeffect of GuiZhiShaoYaoZhiMuTang for treating adjuvant-induced arthritis rat and theby-effect.

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Rats with CHF were divided randomly into two groups, one group was injected dexamethasone(1mg /kg) intramuscularly at the first and forth day respectively,the other group was injected equi-volume saline likewise, and observing the changes of symptoms of rats with CHF; measuring rats hemodynamics index including blood pressure, heart rate and left ventricular end-diastolic pressure 4 days later. Part 2: To measure every group rats myocardialα1亚洲欧美日本一区二区综合、β1、β2亚洲欧美性爱片、β3-AR density by immunohistochemical method ,and to understand the AR density changes .Taking the first part rats'myocardium tissues for testing myocardialα1中文字幕中日韩欧美一区、β1、β2中文字幕第几页、β3-AR density and 5 normal rats for normo-contrast group and taking gray scale of slice as AR density. Part 3: The study enrolled 35 patients with DCHF. Patients were included if they had orthopnea and refractory edema due to acute DCHF that was severe and had taken more 1 week hospitalization and intravenous therapy in addition to diuretics.

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Results:(1)Among 40 rats, 36 rats were successfully established and the rate of success is 90 percent;(2)All the successfully established models demonstrated polydipsia, polyuria and the body weight was not increased. 6 rats suffered cataract after 3 months, and 4 rats died in 6 months;(3)There was an approximately 61% loss of retinal ganglion cells in the central retina and the thickness of retina thinner under microscope ( P 0.01 ).(4) Electroscope changes include the thinner of retina, disorganization of the membranous disc of the rod cells and the thickness of basal membrane of vessal.(5)In normal group, 1 month dibetes mellitus and 1 month treatmen group, there was no expression of ERK1/ERK2 on the retina tissues. In 3 month diabetes mellitus group, the expression of ERK1/ERK2 was positive.

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The SMMC-7721 hepatoma cell line of experimental groups and control groups were cultured with the addition of sodium selenite, potassium iodide or triiodothyronine (T3). The cell quantity was checked by the MTT assay , and the cell cycle was observed by flow cytometry and levels of AFP and ALB in the culture solution were determined by RIA.The SD rats were divided into positive control group, negative control group, T3 group and mixture group (the mixture of sodium selenite, KI and T3). After two weeks of the administration of sodium selenite、 KI and T3,Walker256 cells were transplanted into the abdominal cavity. The administration was kept on for another two weeks, then observed the morbility and mortality of the rats with ascites carcinoma. At the end, the ascites were for the cells counting and the cell cycles analysising.

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This research was composed of three parts:in the first part, we observed the effect of CTA on small intestine peristalsis;in the sencond part, we observed the effect of sweet stimulability and bitter stimulability on discharge of vagus efferent never after CTA;in the three part,effect of CTA on c-Fos expressions in the brain nerve nucleus was observed.

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Each male ratwas paired with 2 female rats in proestrus when administration terminated. Femalerats were examined the next morning for the presence of sperm in vaginal smears andunderwent a cesarean section on day 13 of gestation. Then the reproductive indiceswere calculated as follows: copulation index, pregnancy index, and fertility index,which can evaluate male fertility directly. The sperm motility and morphology wereevaluated by computer-assisted sperm analysis, moreover, the caudaepididymal sperm ultramicrostructure were analyzed by transmission electronmicroscope, which can evaluate the influence on sperm maturation bydutasteride ; sperm survival rate was assessed by SYBR-14 and propidium iodidefluorescent staining; serumal dihydrotestosterone and testosterone of ratswere detected by enzyme-labeled immunoassay, which can evaluate the effecton androgenic levels; the weights of testes and epididymides, were determined byprecise electronic balance; histological examination of above tissues were evaluatedby HE staining and TEM, which can evaluate treatment on histology of reproductiveorgans by dutasteride.

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The effects and mechanism of GABAergic neurons, NOergic neurons, opioid peptide and cyclic adenosine monophosphate in the nucleus reticularis thalami on sleep-wakefulness cycle of rats and the effects and mechanism of the 5-HTergic nerve fibers project from the nucleus raphes dorsalis to RT on sleep-wakefulness cycle of rats were investigated with the methods of brain stereotaxic, nucleus spile, microinjection and polysomngraphy.1. The effects of GABAergic neurons in RT on sleep-wakefulness cycle of rats1.1 Microinjection of 3-mercaptopropionic acid (3-MP, a kind of glutamate decarboxylase inhibitor) into RT. On the day of microinjection, sleep only decreased a litter. On the second day, sleep marked decreased and wakefulness marked increased. On the third and fourth day, sleep and wakefulness stages resumed to normal.1.2 Microinjection of gamma-amino butyric acid (GABA 1.0μg) into RT enhanced sleep and reduced wakefulness compared with control; while microinjection of L-glutamate (L-Glu, 0.2μg) decreased sleep and increased wakefulness; microinjection of bicuculline (BIC, 1.0μg), a GABAA receptor antagonist, enhanced wakefulness and reduced sleep; microinjection of baclofen (BAC, 1.0μg), GABAB receptor agonist, had the same effects as GABA.2. The effects of NOergic neurons in RT on sleep-wakefulness cycle of rats2.1 Microinjection of L-arginine (L-Arg, 0.5μg) into RT decreased sleep compared with control, but there were on statistaical difference between L-Arg group and control; while microinjection of sodium nitroprusside (SNP, 0.2μg), a NO donor into RT, sleep marked decreased and wakefulness marked increased. Microinjection of nitric oxide synthase inhibitor, N-nitro-L-arginine (L-NNA, 2.0μg) into RT enhanced sleep and reduced wakefulness.2.2 After simultaneous microinjection of L-NNA (2.0μg) and SNP (0.2μg) into RT, SNP abolished the sleep-promoting effect of L-NNA compared with L-NNA group; after simultaneous microinjection of L-NNA (2.0μg) and L-Arg(0.5μg) into RT, we found that L-NNA could not blocked the wakefulness-promoting effect of L-Arg.3. The effects of opioid peptide in RT on sleep-wakefulness cycle of rats3.1 Microinjection of morphine sulfate (MOR, 1.0μg) into RT increased wakefulness and decreased sleep compared with control; while microinjection of naloxone hydrochloride (NAL, 1.0μg), the antagonist of opiate receptors, into RT, enhanced sleep and reduced wakefulness.3.2 After simultaneous microinjection of MOR (1.0μg) and NAL (1.0μg) into RT, the wakefulness-promoting effect of MOR and the sleep-promoting effect of NAL were not observed compared with control.4. The effects of cAMP in RT on sleep-wakefulness cycle of rats Microinjection of cAMP (1.0μg) into RT increased sleep and decreased wakefulness compared with control; microinjection of methylene blue (MB,1.0μg) into RT enhanced sleep and reduced wakefulness compared with control.5. The effects of the 5-HTergic nerve fibers project from DRN to RT on sleep-wakefulness cycle of rats5.1 When L-Glu (0.2μg) was microinjected into DRN and normal sodium (NS,1.0μg) was microinjected into bilateral RT. We found that sleep was decreased and wakefulness was increased compared with control; when L-Glu (0.2μg) was microinjected into DRN and methysergide (MS,1.0μg), a non-selective 5-HT antagonist, was microinjected into bilateral RT, We found that sleep was enhanced and wakefulness was reduced compared with L-Glu group.5.2 When p-chlorophenylalanine (PCPA, 10μg) was microinjected into DRN and NS (1.0μg) was microinjected into bilateral RT, We found that sleep was increased and wakefulness was decreased compared with control; microinjection of 5-hydroxytryptaphan (5-HTP, 1.0μg), which can convert to 5-HT by the enzyme tryptophane hydroxylase and enhance 5-HT into bilateral RT, could block the effect of microinjection of PCPA into DRN on sleep-wakefulness cycle.

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The animal model of this method system have, the characteristics of the high fat, high leptin, high Ins, and match with the characteristic of clinical and the pathologic of simple obesity. This studies showed: The acupuncture can obviously reduce the vaule of intaking food and water of the fat rats, and lower the body weight and fat; The acupuncture can urge the diameter, area, physical volume of white fat cell contract, Brown fat tissue of fat rat group surroundings vascular containing large quantity of white fat cell, the acupuncture group was less. this showed: The acupuncture enhanced the white fat cell metabolism, and promotesed the white fat cell to convert to brown fat cell; The frequency of spontaneous electric of nerve cell of pvn of the fat rat is high, the acupuncture counld depress the spontaneous electric of nerve cell of pvn, This showed: the acupuncture can depress the appetite passing to adjust function of pvn nerve; The monoaminergic neurotransmitter of inside of the fat rat brain is disfunction, the acupuncture can rectify this disfunction, and adjust level of 5-HT, and affect appetite, and improve metabolism; The level of leptin and Ins of fat rat increased high, The level of it of acupuncture group decreased, compared with the fat group p.001, This showed: the fat rats have the defeat of leptin and Ins in the body, The acupuncture can lowering this defeat, The level of leptin and Ins of the fat rat in the brain is less than the normal group, The level of leptin of the acupuncture group is high than the fat group, This showed: the acupuncture can increase the transportation of leptin from blood to brain.

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ACTH：大鼠促腎上腺皮質(zhì)激素

大鼠促生長激素釋放激素(GRH) 大鼠促腎上腺皮質(zhì)激素(ACTH) 大鼠促卵泡素(FSH) 大鼠雌二醇受體(ER) 大鼠心鈉肽Pro-ANP(1-98) 大鼠大內(nèi)皮素(BigET) 大鼠降鈣素(CT) 大鼠骨鈣素(OT)ELISA Kit 大鼠促甲狀腺素(TSH) 大鼠皮質(zhì)醇(C

Rat Azidothymidine,AZT ELISA Kit：大鼠疊氮胸苷(AZT)ELISA試劑盒

大鼠補(bǔ)體蛋白3(C3)ELISA試劑盒 Rat Complement 3,C... | 大鼠疊氮胸苷(AZT)ELISA試劑盒 Rat Azidothymidine,AZT ELISA Kit | 大鼠高靈敏度促甲狀腺激素(U-TSH)ELISA試劑盒 Rat ultrasensitive thyroid-stimulating ho...

(CORT)ELISA Kit rat Corticosterone,CORT ELISA Kit：大鼠皮質(zhì)酮/腎上腺酮

RC02100B 大鼠皮質(zhì)酮/腎上腺酮(CORT)ELISA Kit rat Corticosterone,CORT ELISA Kit | RC02101B 大鼠內(nèi)皮素1(ET-1)ELISA Kit rat Endothelin 1,ET-1 ... | RC02102B 大鼠血管舒緩激肽(BK)ELISA Kit Rat bradykinin,BK ...

CT：大鼠降鈣素

大鼠促生長激素釋放激素(GRH) 大鼠促腎上腺皮質(zhì)激素(ACTH) 大鼠促卵泡素(FSH) 大鼠雌二醇受體(ER) 大鼠心鈉肽Pro-ANP(1-98) 大鼠大內(nèi)皮素(BigET) 大鼠降鈣素(CT) 大鼠骨鈣素(OT)ELISA Kit 大鼠促甲狀腺素(TSH) 大鼠皮質(zhì)醇

cytochrome C：大鼠細(xì)胞色素

CTLA-4 大鼠細(xì)胞毒T淋巴細(xì)胞相關(guān)抗原4 | Cytochrome-C 大鼠細(xì)胞色素C | Emap-2 大鼠內(nèi)皮單核細(xì)胞活化多肽2

(TAFI)ELISA Kit Rat Thrombin activatable fibrinolysis inhibitor,TAFI ELISA Kit：大鼠纖溶抑制因子/凝血酶激活的纖溶抑制物

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rat,roof：房頂(大)鼠

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Rat rudimental bovine serum albumin check-up ELISA Kit：大鼠的牛血清白蛋白殘留檢測ELISA試劑盒

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Rattus norvegicus：挪威(大)鼠,棕色(大)鼠,褐鼠

\\"鼠屬\\",\\"Rattus\\" | \\"挪威(大)鼠,棕色(大)鼠,褐鼠\\",\\"Rattus norvegicus\\" | \\"房頂(大)鼠\\",\\"Rattus rattus\\"

Rattus rattus：房頂(大)鼠

\\"挪威(大)鼠,棕色(大)鼠,褐鼠\\",\\"Rattus norvegicus\\" | \\"房頂(大)鼠\\",\\"Rattus rattus\\" | \\"輕麻醉\\",\\"rausch\\"