Methods On the basis of bilirubin-induced encephalopathy model in guinea-pig, 10 μg/g of GAPA was injected intraperitoneal.

在制作膽紅素腦病動物模型基礎上予GAPA干預，觀察對照組538在线在视频在线、模型組四位少妇黑店精油按摩、模型干預組新生豚鼠（每組10只）干預后4、8 h腦組織超微結構人人爱人人操怕怕、腦組織ATP含量與含水量97精品国产91久久久久久久。

The GC contents of the entire region of the GapA cDNA (64.2%) were similar to the average GC content (65.2%) which was obtained by Porphyra EST analysis.

條斑紫菜GapA的cDNA序列中GC含量很高(64.2％)，與紫菜EST分析中得到的結果(65.2％)近似曰批视频观看全过程120秒XXXX。

Results The brain ATP production in bilirubin induced neurotoxicity guinea-pig decreased within 4 hours after exposure to bilirubin, and secondary brain edema could be found 8 hours later; GAPA could prevent the secondary brain edema effectively ,but could not prevent the ATP decreasing significantly.

沉積于腦組織膽紅素可抑制神經(jīng)元能量代謝久久永久免费在线，致腦細胞水腫；興奮性氨基酸受體拮抗劑GAPA可減輕腦水腫欧美精选三区，但不影響能量代謝變化超黄色毛片久国产，其作用環(huán)節(jié)介于能量代謝變化與腦水腫之間。

Methods On the basis of bilirubin induction and NMDA receptor an-tagonist GAPA administration in guinea pigs. The brain extracellular EAA neurotransmitter was ex-tracted with microdialysis and analysed with HPLC; then, brain tissue ATP was measured by fluores-cence assay and intracellular Ca2 + was measured by fluorescence imaging analysis.

制作膽紅素腦病動物模型基礎上gzdaqm.com，在體腦內(nèi)微透析提取興奮性氨基酸神經(jīng)遞質色污污网站，HPLC檢測天冬氨酸、谷氨酸欧美精品三区、甘氨酸www.A黄色网站，并給予模型動物NMDA受體拮抗劑GAPA干預，電鏡檢測腦組織超微結構看黄网站网址免费，熒光檢測腦組織ATP與細胞內(nèi)Ca2+含量欧洲美洲精品一区二区三区。

Results ATP andEAA Neurotransmitter Gly were greatly increased in the bilirubin induced neurotoxicity brain; neuralcytosolic Ca2 + in the bilirubin precipitated brain tissue was significantly more than that in the controlgroup; NMDA receptor antagonist GAPA could significantly decrease the cytosolic Ca2+ overload.

結果 膽紅素毒性腦組織ATP含量顯著降低，細胞外Gly與細胞內(nèi)Ca2+顯著升高亚洲一区二区三区羽田爱；GAPA不影響膽紅素毒性腦組織ATP含量美国一级黄片的黄色美国的现在能放出来的，但可顯著降低細胞內(nèi)Ca2+，減輕膽紅素毒性腦水腫狠狠色视频一区二区。

EAA neurotransmitter Gly increased the sensitivity of the ligand Asp and Glu to theNMDA receptor, thus increased the NMDA receptor activity and cytosolic Ca2 + in the bilirubin precipi-tated brain tissue. NMDA receptor antagonist GAPA could prevent the cytosolic Ca2 + overload, and re-lieve brain edema.

NMDA受體過度活化致細胞內(nèi)Ca2+超載參與介導膽紅素神經(jīng)毒性一级黄色视频免费下载观看真人视频，NMDA受體拮抗劑可望成為膽紅素神經(jīng)毒性防治的重要途徑。