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英語人>網(wǎng)絡(luò)例句>null function 相關(guān)的網(wǎng)絡(luò)例句
null function相關(guān)的網(wǎng)絡(luò)例句

查詢詞典 null function

與 null function 相關(guān)的網(wǎng)絡(luò)例句 [注:此內(nèi)容來源于網(wǎng)絡(luò)伊人超碰完整版一区二区,僅供參考]

In order to detect the nonlinear determinism, the method of surrogate is employed. The null hypothesis that a real system response is from a stationary Gaussian linear process distorted by an invertible, static, nonlinear measurement function is made and tested.

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In this thesis, we find that dlg is indispensible in the establishment of anterior-posterior and dorsal-ventral polarity of drosophila oocyte. Removal of Dlg function from the posterior follicle cells using the FLP/FRT system leads to disruption of oocyte skeleton reconstruction that is elicited by the failure of those posterior cells to differentiate normally in mid-oogenesis. We demonstrate that abnormity of Notch, JAK-STAT and EGFR signal pathway in dlg mutants contributes to this aberrant differentiation. dlg null mutant also blocks the normal differentiation of two groups of anterior follicle cell-stretched cell and centripetal cell, but not border cell, with a lower penetrance. However unlike the result in posterior follicle cells, Notch and JAK-STAT signaling are both undisrupted in all mutant anterior follicle cells, implying other fate determinants may be involved.

我們的研究發(fā)現(xiàn),后端濾泡細(xì)胞中的Dlg在果蠅卵子發(fā)生中期卵母細(xì)胞前后軸和背腹軸建立過程中也是必須的久久99看黄毛片免费,PFC中dlg完全缺失型突變引起PFC的分化異常2014Av天堂网旧版,導(dǎo)致卵子發(fā)生中期卵母細(xì)胞骨架重組異常,Stau日韩精品美女自拍网页在线观看免费、Grk等極性決定蛋白定位錯誤色综合色欲色综合色综合色综合。dlg突變阻礙了Notch、JAK-STAT91色偷偷色噜噜狠狠网站、EGFR等調(diào)節(jié)PFC分化的信號通路的激活1024色婬免费视频。dlg突變的PFC也沒有獲得前端濾泡細(xì)胞命運(yùn)一级无遮挡真人毛片A片。dlg突變不影響前端濾泡細(xì)胞群中邊界細(xì)胞的分化无码啊片,但是在一定程度上影響伸展細(xì)胞和向心細(xì)胞的分化真人一收a片免费视频女白领,并且這種影響不依賴于前端濾泡細(xì)胞Notch或JAK-STAT信號激活的異常。

Background— Protein kinase A signaling has long been known to play an important role in cardiac function. Dysregulation of the protein kinase A system, caused by mutation of the protein kinase A regulatory subunit gene PRKAR1A, causes the inherited tumor syndrome Carney complex, which includes cardiac myxomas as one of its cardinal features. Mouse models of this genetic defect have been unsatisfactory because homozygote null animals die early in development and heterozygotes do not exhibit a cardiac phenotype.

背景——蛋白激酶A信號傳導(dǎo)在心功能中發(fā)揮了重要作用早已為人們所知蛋白激酶A調(diào)節(jié)亞基因PRKAR1A的變化引起蛋白激酶A系統(tǒng)失調(diào)欧美久久久久久,導(dǎo)致了遺傳性卡尼復(fù)合腫瘤綜合癥五月丁香六月婷婷综合,其中包括的心臟粘液瘤作為它的一個主要特征此種遺傳缺陷的小鼠模式仍然不能令人滿意,因其純合體空白動物在發(fā)展中早期死亡一级欧美片一级日韩美片AAA,而雜合體不能顯示心臟表型

Perutz M F Structure and Function of Haemoglobin Journal of Molecular Biology , 1965,null

王耀萍%潘冀%牛秀田我國第二次空間蛋白質(zhì)晶體生長實驗中國科學(xué)C輯一区二区三区无码免费视频, 1996,2

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