The recovery of blood circulation of ischemia semidarkness region, improvement of partly hypoxia state and prevention of more serious of impaired are key point in the stroke treatment. This experiment is designed to observe the blood vessel regeneration of ischemia injured region, and observe the variance of partly PCNA and VEGF expression. Combine with limbs functional recovery, we validate the therapeutical effect of acupuncture and investigate the possible mechanism of action.

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Boosts warm water bag of wound healing tepidity being able to stimulate the effect organizing regeneration and having lighten pain and reinforce organization nutrition , large amount of seriflux exudate increases by , can help to eliminate pathological mechanism outcome when the wound thinking that the tepid effect administers medicine to bring out the cold in the body,; Another , fever may distend the blood-vessel , blood vessel Tongtou sex strengthens, snapout beneficial to organizing supersession outcome and the sorption to nutrient substance, restrain development of inflammation , promote a wound healing.

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Our data indicate that mitochondria might be the most vunerable organelle to areca alkaloids . Furthermore ,the occurrence of vesiculation seemed to be PCE-specific ,In conclusion ,at the concentration of MSD , PCE of areca fruit is able to induce a series of reversible changes in the cytoplasm of treated cells .

本文的結(jié)論﹐乃是利用檳榔果實的PCE及兩種檳榔堿﹐在 MSD的濃度下﹐我們可以誘發(fā)一系列可逆性的細(xì)胞變化可變誘發(fā)﹐此一可逆性的特質(zhì)可能牽涉到PCE可先破壞或停止細(xì)胞的合成作用99国产网站，誘發(fā)細(xì)胞解毒作用﹐再激活細(xì)胞再生的過程或機轉(zhuǎn)情侣自拍网。

Objective to evaluate the functional recovery of acute spinal cord injuried rats treated with exogenous wnt-3a signal protein administration and to explore its mechanism.methods moderate spinal cord contusion injury was made in 40 adult sprague dawley rats at t10.twenty rats served as contusion controls(group 1).twenty rats were treated with wnt-3a for three days after injury (group 2).the functional recovery of the rats was observed through basso,beattie,bresnahan open field locomotor score.rats were killed at 14 or 28 days after injury,then spinal cords were removed for histopathological examinations,and the expression of the bromodeoxyuridine plus neural cell markers was stained with immunohistochemical method.results rats of two groups receiving a contusive injury recovered substantial function within 1 week.by 28 days,rats in groups 2 scored 7.0 points better on the bbb scores than rats in group 1 group 2=16.94,after 28 days vs.

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The reason is that warm has functions of stimulating tissue regeneration, alleviating pain and increasing tissue nutrition, when warm acts on surface wound, serous effusion increases, which helps to eliminate pathological products; warm can make blood vessel dilative and enhance vascular permeability, which is propitious to discharge tissue metabolites, absorb nutriment, restrain development of inflammation and advance the healing.

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Correlation analyses on the contents and ratios of endogenous hormones with selfing fertilities showed that selfing fertility was not determined only by one single hormone level, but partially affected by the joint action or balance of several hormones in Nongken58S original and untransformed regenerant controls under long-day. Our results did not support different already proposed hypotheses on one single hormone action in fertility expression of PSGMS rice.

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Gelatin microsphere is firstly made by encapsulating bFGF into gelatin by special manufacture process, then this microsphere was combinated into PLGA conduit or PLGA film.when used, we need only pack the injury peripheral nerve, injury spinal cord, wounded pars encephalic with the PLGA conduit. The conduit or film can slowly release buff which would be absorbed by around tissues.bFGF was already proved to be nutritious to the regeneration and alive of neuron and protect injury neuron.bFGF can also promote morphogenesia and division of nerve cell which would raise the survival rate of neuron, guide the generation of axon etc.bFGF is one of the important factors of sustaining the normal survival of cholinergic nerve.

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