Results ⑴ The cell proliferation was inhibited by TET. It showed thatdivision index and cell number during G0-G1 were decreased, while S and G2-M phases were lengthened with dosage and time dependency in dosages of TET from 2mg/L to 8mg/L; besides, oval-shap cells, karyopyknosis and fragmentation were observed under light microscope. The inhibitory effect in T2 group was statistically similar to that in triamcinolone.

結(jié)果⑴ TET在2mg/L~8mg/L之間呈劑量及時間依賴性地抑制瘢痕成纖維細(xì)胞的增殖性爱小视频欧美，使生長曲線不同程度的壓低色黄乱婬伦免费网站耙、分裂指數(shù)降低日韩三级AV网站、G0-G1期細(xì)胞數(shù)減少www.AAA片黄色网址、G2-M期增多、S期與G2-M期延長日韩一级免费视频网站，光鏡下見細(xì)胞突起變短亚洲免费播放片，細(xì)胞由梭形變成橢圓形，核固縮又粗又硬又大欧美69a√、碎裂。P.C的改變近似于T2組(4mg/L TET)爱情岛论坛自拍亚洲品质福利。

Its characteristic changes are monocellular occurrence,pyknosis,karyorrhexis and the formation of apoptotic body.

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The proliferating cells were mainly distributed in the peri-chondrium and the peripheral zone of tracheal cartilage. The senescent chondrocytes were distributed in the central zone whereapoptotic morphological characteristics obviously appeared, such as pyknosis, karyorrhexis.

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Results HE staining showed that sulindac could inhibit the growth of tumor cells, with the morphological presentations as apoptosis and necrosis including pyknosis, karyorrhexis and chromatin crescent formation, etc. By electron microscopy, cells also had apoptotic characteristics including chromatin crescent formation and apoptotic body. Immunohistochemistry and Western-blot assay results showed that 48 hours after sulindac administration, the expression of NF-κB, bcl-2 and bax gradually decreased, while the level of bax protein gradually increased, in a dose dependence manner.

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Typical characteristics of apoptotic cells as membrane blebbing, chromatin pyknosis, karyorrhexis, and apoptotic body were seen under electromicroscope. Apoptosisspecific DNA ladder bands from SGC-7901 cells were visualized with electrophoresis. Hypodiploid apoptotic peak was observed before G1 phase by flow cytometry analysis.

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When the fault throw is relatively small, the shale gouge ratio and clay smear potential are relatively large and shale smear is strong. In addition, the coal layers on both sides of the fault are mainly juxtaposed against shale. Therefore, shale smear and juxtaposition play a key role in the sealing. In contrast, when the fault throw is relatively large and shale smear is relatively weak, juxtaposition is unfavorable for sealing. However, the mylonite resulted from strong cataclasis can act as a seal of the coals. The cementation resulted from underground water activity is of great significance to the sealing ability of the fault. Mylonitic coal layers resulted from faulting occur on both sides of the fault. They are unique reservoirs with low permeability and high displacement pressure and their scales depend, to some extent, on the fault throw, fault properties, the hanging wall and footwall.

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After 48h exposure to 50mg/L ox-LDL, under the fluorescent microscope partial cells Showed typical apoptotic morphological changes such as nacarat fluorescent body shrinking and fragment of the nucleus. At the same time, the cellular cholesterol content also increased obviously, and many lipid droplets accumulated in cells.

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The fundings in-operation were:complicated spinal canal stenosis was in9cases,luted ligament pachynsis in45cases,some tissue bulged into vertebral canal and had fragmentation in9cases,in one case calcifieation appeared.The follow-up period was six months to one year,all patients operated on were lived a normal life.

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A high failure rate of stand-alone, short-segment pedicular fixation in the setting of thoracolumbar burst fractures prompted the development of the load-sharing classification system of thoracolumbar fractures, which allowed guidance in understanding the potential for failure of posterior instrumentation alone in the setting of significant disruption of the anterior vertebral body.

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