The tuberculosis patients with proteinic malnutrition emerge medicinal liver function injures easily in the treatment process, so that we should monitor the clinical treatment for their liver function changes and pay attention to the improvement in their nutritional conditions.

在肺結(jié)核并蛋白質(zhì)營養(yǎng)不良的患者治療過程中乱淫网站，較容易出現(xiàn)藥物性肝損害pp.94在线免费。因此在臨床治療上亚洲一区二区三区a片，應(yīng)注意監(jiān)測其肝功能變化以及注意改善其營養(yǎng)狀態(tài)一级黄片儿一级大a黄片啪，減少藥物性肝損害發(fā)生91精品夜夜夜一区二区。

A total of 769 pulmonary tuberculosis patients without proteinic malnutrition, who were admitted to the hospital during the same periods new were selected into control group.

結(jié)果 合并蛋白質(zhì)營養(yǎng)不良初治肺結(jié)核患者AAAAAAAAA欧美黄色视频，在治療第1個月出現(xiàn)藥物性肝損害較對照組高(χ^2=4605， P=0.031亚洲天堂无码人妻在线影院， RR=1.5)沙特毛片；在治療第1個月的藥物性肝損害發(fā)生率污污网址免费观看，較治療第2個月高(χ^2=15.183， P=0.001)cao女人。

The mean retention time in liver and lung was longer and was eliminated more slowly than other organs. It suggested that there was a correspondence relationship between OADHPS and its hepato-protection. After oral administration, 14. 8％ of OADHPS was excreted as protype. It was assumed that 10.0％ of OADHPS was metabolized in vivo whereas 75. 2％ of OADHPS destroyed in gastrointestinal tract.

藥物在肝欧美黄色一级A片视频、肺等組織中的平均滯留時間較長，消除相對較慢久久免费看A级毛片色欲，這恰好與齊墩果酸具有的較強(qiáng)的保肝作用相呼應(yīng)三级中文字幕无码视频。24h尿中原型藥物排泄量占給藥量的3.21％，結(jié)合絕對生物利用度為21.7％思思热在线观看视频，推測被吸收的藥物中約14.8％以原型排泄视色,视色,视色影院视色影库。

The percutaneous pulsive current stimulation of hepatic region in exercise-induced fatigue rats may improve the activity of antioxidant enzymes in liver, promote free radical-scavenging and hepatic glycogen synthesis, delay the occurrence of exercise-induced fatigue, and promote the recovery from exercise-induced fatigue.

脈沖電流經(jīng)皮刺激運(yùn)動疲勞大鼠肝區(qū)可提高肝臟抗氧化酶的活性，促進(jìn)機(jī)體自由基的消除及肝糖原的合成日韩欧美图片，具有延緩運(yùn)動疲勞發(fā)生7色在色在线视频、促進(jìn)運(yùn)動疲勞恢復(fù)的作用。

Results The bletilla colloid was a homogenous viscous colloid whose relative viscosity was 2324.6 mm2/s. It was easily injected through 4-F catheter and hyperattenuating under fluoroscopy, meanwhile, with good histocompatibility and hemo-compatibility, without pyrogenetic response and toxicity. In vitro, the mixture of bletilla colloid and MMC did not produce separation and suspention phenomena but released 50%of MMC at 1.8h and 100%at 3.4h. The bletilla colloid mainly embolized peripheral arteries, maintaining occlusion for 5 weeks and without formation of collateral circulation. The injuries of normal hepatic tissues were slight, without hepatic cytonecrosis.

結(jié)果 (1)白芨膠為一種均質(zhì)的膠狀物日日射日日插日日干，有一定粘滯性欧美成免费黄色视频，相對粘度2 324.6 mm2/s，不透X線在线观看免费毛片高清，易經(jīng)4F導(dǎo)管注射日韩天堂免费视频在线；(2)白芨膠具有良好的血液相容性和組織相容性，無致熱原性及毒性作用黄色网站动；(3)白芨膠-絲裂霉素C體外混合后中国区第一黄色网，無分層及懸浮現(xiàn)象，釋放MMC 50%約需1.8小時这里全是是精品99，100%需3.4小時美国黄色视频黄色视频聊天；(4)白芨膠主要栓塞末梢小動脈，栓塞牢靠爱情岛论坛福利永久首页，維持時間可達(dá)5周超碰91成人人做人人爱，不易形成側(cè)支循環(huán)；(5)白芨膠栓塞對正常肝組織損傷輕微色91视频综合，未出現(xiàn)肝細(xì)胞壞死亚洲爆乳少妇无码激情。

Previous studies have found the major metabolic way of clivorine in male rat liver microsomes was to form pyrrolic metabolites. The above results suggested that there is sexual difference of metabolism in rats. The major in vitro metabolic way of clivorine in guinea pig was hydrolysis.

FMO的特異性抑制劑methimazo1e可以顯著地抑制〓的形成，但對〓的形成無明顯的抑制作用91cao，且〓在肝微粒體中的形成為NADPH非依賴性亚洲天堂免费看，上述結(jié)果提示參與〓和〓代謝的酶分別為肝微粒體中的水解酶和黃素單氧化。

The major in vitro metabolites of clivorine found in the incubation mixture are two non-pyrrolic metabolites(M1 and M2) and the formation of hepatotoxic pyrrolic metabolites from clivorine is only the minor in vitro metabolic pathway of clivorine in female rats.

山岡橐吾堿在雌性大鼠肝微粒體內(nèi)的主要代謝物為兩個非吡咯代謝物M1和M2欧美性爱视频一日本高清视频。與雄性大鼠不同精品黄色视频在线，生成肝毒性的吡咯代謝物為其次要的代謝途徑。

The rat im-munohepatic fibrosis model was made by intraperitoneal injection of porcine serum and treated with rhizoma sparganii and radices zedoariae. The ALT,GGT,TP, ALb, A/G, IVC,LN,HA and the pathological change of the liver were observed.

以腹腔注射豬血清制備大鼠肝纖維化模型，以ALT自拍偷拍午夜视频，GGT三级特大黄片视频影院大国产农村，TP，Alb九九自拍偷拍，A/G比值欧美一级特黄大片做受在线观看人人操，ⅣC，LN日本人男女乱伦发生性关系视频。，HA及肝臟組織病理變化為指標(biāo)jizzjizzjizz亚洲日本，觀察三棱、莪術(shù)對大鼠免疫性肝纖維化的影響人人摸人人色人人操。

The FDA did not relabel Ketek to indicate its possible severe hepatotoxicity until 16 months after the first liver-failure cases became public.

直到第一例肝衰竭病例公開后的16個月www.色棋棋.com，F(xiàn)DA才重新在Ketek的標(biāo)簽中注明其可能具有嚴(yán)重肝毒性。

Bilberry extract was oral administrated to mice for 5d,which remarkedly decreased ALT activity,and protect the liver damage induced by oxidative stress.

給予歐洲越橘花色苷提取物可以明顯降低拘束負(fù)荷誘發(fā)小鼠血漿ALT活性升高网站男人天堂，清除體內(nèi)自由基国产大片精品黄页免费看，恢復(fù)肝組織GSH和vitaminC水平，降低肝組織過高的MDA和NO水平观看欧美黄色网站。